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W421501

酪胺

Name: 酪胺
Brand: ALDRICH

98%, FG

别名:

2-(4-羟基苯基)乙胺, 4-(2-氨乙基)苯酚, 4-羟基苯乙胺

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关于此项目

线性分子式:

HOC₆H₄CH₂CH₂NH₂

化学文摘社编号:

51-67-2

分子量:

137.18

Flavis number:

11.007

PubChem Substance ID:

329830683

UNSPSC Code:

12164502

FEMA Number:

4215

NACRES:

NA.21

EC Number:

200-115-8

MDL number:

MFCD00008193

Beilstein/REAXYS Number:

1099914

Organoleptic:

meaty; phenolic

Grade:

Biological source:

synthetic

Food allergen:

no known allergens

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属性

biological source

synthetic

Quality Level

300, 400

grade

reg. compliance

EU Regulation 1334/2008 & 872/2012, FDA 21 CFR 110

assay

98%

bp

175-181 °C/8 mmHg (lit.)

mp

160-162 °C (lit.)

application(s)

flavors and fragrances

documentation

see Safety & Documentation for available documents

food allergen

no known allergens

organoleptic

meaty; phenolic

SMILES string

NCCc1ccc(O)cc1

InChI

1S/C8H11NO/c9-6-5-7-1-3-8(10)4-2-7/h1-4,10H,5-6,9H2

InChI key

DZGWFCGJZKJUFP-UHFFFAOYSA-N

说明

Biochem/physiol Actions

可以进入儿茶酚胺能末梢并作为伪递质释放。

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pictograms

GHS07

signalword

Warning

hcodes

H315,H319,H335

pcodes

P261 - P264 - P271 - P280 - P302 + P352 - P305 + P351 + P338

Hazard Classifications

Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

target_organs

Respiratory system

存储类别

11 - Combustible Solids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

dust mask type N95 (US), Eyeshields, Gloves

法规信息

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历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Neurovascular dissociation with paradoxical forearm vasodilation during systemic tyramine administration.

Giris Jacob et al.

Circulation, 107(19), 2475-2479 (2003-04-23)

Despite the widespread use of tyramine as a pharmacological tool to assess the effects of norepinephrine release from sympathetic nerve terminals, its vascular effects are not adequately characterized. In particular, previous results indicate that intravenous tyramine produces little if any

Selective modulation through the glucocorticoid receptor ameliorates muscle pathology in mdx mice.

Tony Huynh et al.

The Journal of pathology, 231(2), 223-235 (2013-06-25)

The over-expression of NF-κB signalling in both muscle and immune cells contribute to the pathology in dystrophic muscle. The anti-inflammatory properties of glucocorticoids, mediated predominantly through monomeric glucocorticoid receptor inhibition of transcription factors such as NF-κB (transrepression), are postulated to

Endothelium- and beta-2 adrenoceptor-independent relaxation of rat aorta by tyramine and certain other phenylethylamines.

D R Varma et al.

The Journal of pharmacology and experimental therapeutics, 265(3), 1096-1104 (1993-06-01)

At concentrations higher than that required to produce maximal vasoconstriction, tyramine caused concentration-dependent relaxation of rat aortic strips contracted maximally by tyramine, norepinephrine, phenylephrine, 5-hydroxytryptamine, prostaglandin F2 alpha, endothelin, angiotensin II and potassium; isoproterenol did not relax potassium-contracted strips. The

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