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Merck
CN

SAB1305560

Anti-MAP Kinase- ERK1/ERK2 Antibody

mouse monoclonal, 784CT7.6.3

别名:

ERK1, MAPK3, Mitogen-activated protein kinase 3, PRKM3

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关于此项目

UNSPSC Code:
12352203
NACRES:
NA.41
Clone:
784CT7.6.3, monoclonal
Species reactivity:
mouse, human
Application:
WB
Citations:
5
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产品名称

MONOCLONAL ANTI-ERK1/2 antibody produced in mouse, clone 784CT7.6.3, IgG fraction of antiserum, buffered aqueous solution

biological source

mouse

Quality Level

antibody form

IgG fraction of antiserum

antibody product type

primary antibodies

clone

784CT7.6.3, monoclonal

form

buffered aqueous solution

mol wt

43136 Da

species reactivity

mouse, human

technique(s)

western blot: 1:1000

isotype

IgG2a

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... MAPK3(5595)

Physical form

Supplied in PBS with 0.09% (W/V) sodium azide

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.


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存储类别

10 - Combustible liquids

flash_point_f

Not applicable

flash_point_c

Not applicable

法规信息

低风险生物材料
常规特殊物品

此项目有



历史批次信息供参考:

分析证书(COA)

Lot/Batch Number

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Yuhang Yuan et al.
Cell cycle (Georgetown, Tex.), 17(11), 1390-1402 (2018-06-29)
Necrotizing enterocolitis (NEC) is a potentially fatal illness in premature neonates. Tumor necrosis factor-α (TNF-α) and autophagy are associated with the pathogenesis of NEC. This study aimed to explore whether TNF-α might regulate apoptosis in neonatal NEC model cells IEC-6
Barathi Rajaraman et al.
Molecular and cellular biochemistry, 456(1-2), 179-190 (2019-02-16)
Hyperglycaemia during pregnancy is the main reason for developing diabetes mediated vascular complications. Advanced glycation end products (AGEs) are formed due to non-enzymatic glycation of proteins, lipids and nucleic acids during hyperglycaemia. It has the potential to damage vasculature by
Davide D'Amico et al.
Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 42(2), 462-472 (2016-08-19)
The inability to properly extinguish fear memories constitutes the foundation of several anxiety disorders, including panic disorder. Recent findings show that boosting prefrontal cortex synaptic plasticity potentiates fear extinction, suggesting that therapies that augment synaptic plasticity could prove useful in



全球贸易项目编号

货号GTIN
SAB1305560-400UL04061836266752