SAB1305560
Anti-MAP Kinase- ERK1/ERK2 Antibody
mouse monoclonal, 784CT7.6.3
Synonym(s):
ERK1, MAPK3, Mitogen-activated protein kinase 3, PRKM3
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About This Item
UNSPSC Code:
12352203
NACRES:
NA.41
Clone:
784CT7.6.3, monoclonal
Species reactivity:
mouse, human
Application:
WB
Citations:
5
Product Name
MONOCLONAL ANTI-ERK1/2 antibody produced in mouse, clone 784CT7.6.3, IgG fraction of antiserum, buffered aqueous solution
biological source
mouse
Quality Level
antibody form
IgG fraction of antiserum
antibody product type
primary antibodies
clone
784CT7.6.3, monoclonal
form
buffered aqueous solution
mol wt
43136 Da
species reactivity
mouse, human
technique(s)
western blot: 1:1000
isotype
IgG2a
UniProt accession no.
shipped in
wet ice
storage temp.
−20°C
target post-translational modification
unmodified
Gene Information
human ... MAPK3(5595)
Physical form
Supplied in PBS with 0.09% (W/V) sodium azide
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Storage Class
10 - Combustible liquids
flash_point_f
Not applicable
flash_point_c
Not applicable
Regulatory Information
低风险生物材料
常规特殊物品
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Yuhang Yuan et al.
Cell cycle (Georgetown, Tex.), 17(11), 1390-1402 (2018-06-29)
Necrotizing enterocolitis (NEC) is a potentially fatal illness in premature neonates. Tumor necrosis factor-α (TNF-α) and autophagy are associated with the pathogenesis of NEC. This study aimed to explore whether TNF-α might regulate apoptosis in neonatal NEC model cells IEC-6
Barathi Rajaraman et al.
Molecular and cellular biochemistry, 456(1-2), 179-190 (2019-02-16)
Hyperglycaemia during pregnancy is the main reason for developing diabetes mediated vascular complications. Advanced glycation end products (AGEs) are formed due to non-enzymatic glycation of proteins, lipids and nucleic acids during hyperglycaemia. It has the potential to damage vasculature by
Davide D'Amico et al.
Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 42(2), 462-472 (2016-08-19)
The inability to properly extinguish fear memories constitutes the foundation of several anxiety disorders, including panic disorder. Recent findings show that boosting prefrontal cortex synaptic plasticity potentiates fear extinction, suggesting that therapies that augment synaptic plasticity could prove useful in
Global Trade Item Number
| SKU | GTIN |
|---|---|
| SAB1305560-400UL | 04061836266752 |